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Author
Stephen J. Elledge
also known as Elledge, Stephen J., S Elledge, S J Elledge
Howard Hughes Medical Institute · Harvard University · Stowers Institute for Medical Research · Brigham and Women's Hospital
About this author
Works
540
Cited by
122,689
h-index
172
i10
378
ORCID ↗
Top papers
The DNA Damage Response: Making It Safe to Play with Knives
Alberto Ciccia, Stephen J. Elledge
·
2010
·
Molecular Cell
↗ 4,283
OA
The DNA damage response: putting checkpoints in perspective
Bin‐Bing S. Zhou, Stephen J. Elledge
·
2000
·
Nature
↗ 3,182
ATM and ATR Substrate Analysis Reveals Extensive Protein Networks Responsive to DNA Damage
Shuhei Matsuoka, Bryan A. Ballif, Agata Smogorzewska, et al.
·
2007
·
Science
↗ 3,055
Sensing DNA Damage Through ATRIP Recognition of RPA-ssDNA Complexes
Lee Zou, Stephen J. Elledge
·
2003
·
Science
↗ 2,655
Mice Lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control
Chuxia Deng, Pumin Zhang, J. Wade Harper, et al.
·
1995
·
Cell
↗ 2,117
OA
Cell Cycle Checkpoints: Preventing an Identity Crisis
Stephen J. Elledge
·
1996
·
Science
↗ 2,042
Longitudinal analysis reveals high prevalence of Epstein-Barr virus associated with multiple sclerosis
Kjetil Bjørnevik, Marianna Cortese, Brian C. Healy, et al.
·
2022
·
Science
↗ 1,929
Dicer is essential for mouse development
Emily Bernstein, Sang Yong Kim, Michelle A. Carmell, et al.
·
2003
·
Nature Genetics
↗ 1,908
Principles of Cancer Therapy: Oncogene and Non-oncogene Addiction
Ji Luo, Nicole L. Solimini, Stephen J. Elledge
·
2009
·
Cell
↗ 1,760
OA
The DNA Damage Response: Ten Years After
J. Wade Harper, Stephen J. Elledge
·
2007
·
Molecular Cell
↗ 1,668
OA
A quantitative atlas of mitotic phosphorylation
Noah Dephoure, Chunshui Zhou, Judit Villén, et al.
·
2008
·
Proceedings of the National Academy of Sciences
↗ 1,625
OA
Chk1 is an essential kinase that is regulated by Atr and required for the G<sub>2</sub>/M DNA damage checkpoint
Qinghua Liu, Saritha Guntuku, Xian-Shu Cui, et al.
·
2000
·
Genes & Development
↗ 1,615
OA