Dennis J. Selkoe

The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics

Alzheimer's Disease: Genes, Proteins, and Therapy

The amyloid hypothesis of Alzheimer's disease at 25 years

Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid β-peptide

Alzheimer's Disease Is a Synaptic Failure

Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo

Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Complement and microglia mediate early synapse loss in Alzheimer mouse models

Advancing research diagnostic criteria for Alzheimer's disease: the IWG-2 criteria

Secreted amyloid β–protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

Two transmembrane aspartates in presenilin-1 required for presenilin endoproteolysis and γ-secretase activity

Aβ Oligomers – a decade of discovery